CDK1/FBXW7 facilitates degradation and ubiquitination of MLST8 to inhibit progression of renal cell carcinoma-Reference-Cited by-同舟云学术

CDK1/FBXW7 facilitates degradation and ubiquitination of MLST8 to inhibit progression of renal cell carcinoma

Published:2021-11-16 Issue:1 Volume:113 Page:91-108
ISSN:1347-9032
Container-title:Cancer Science
language:en
Short-container-title:Cancer Science

Author:

Zhang Encheng¹,Chen Siteng¹,Tang Heting¹,Fei Cheng¹,Yuan Zhihao¹,Mu Xingyu¹,Qin Yan¹,Liu Haixia²,Fan Yu³,Tan Mingyue⁴,Wang Xiang¹^ORCID

Affiliation:

1. Department of Urology Shanghai General Hospital Shanghai Jiao Tong University School of Medicine Shanghai China

2. State Key Laboratory of Genetic Engineering Collaborative Innovation Center for Genetics and Development School of Life Sciences Fudan University Shanghai China

3. Department of Renal Transplantation Xiang'an Hospital of Xiamen University Xiamen China

4. Department of Urology Shuguang Hospital Shanghai University of Traditional Chinese Medicine Shanghai China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Cancer Research,Oncology,General Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/cas.15188

Reference35 articles.

1. mTOR Signaling in Growth, Metabolism, and Disease

2. Targeting the PI3K pathway in cancer: are we making headway?

3. Targeting the mTOR regulatory network in hepatocellular carcinoma: Are we making headway?

4. Enhanced mLST8 Expression Correlates with Tumor Progression in Hepatocellular Carcinoma

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