Ischemic post-conditioning protects brain and reduces inflammation in a rat model of focal cerebral ischemia/reperfusion
Author:
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience,Biochemistry
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1471-4159.2008.05276.x/fullpdf
Reference48 articles.
1. Reperfusion Injury: Demonstration of Brain Damage Produced by Reperfusion after Transient Focal Ischemia in Rats
2. The Role of Inflammation and Cytokines in Brain Injury
3. Inflammatory Mediators and Stroke: New Opportunities for Novel Therapeutics
4. Inflammation and acute stroke
5. PI 3-kinase regulates the mitochondrial transition pore in controlled reperfusion and postconditioning
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