Genetic inactivation of p62 leads to accumulation of hyperphosphorylated tau and neurodegeneration-Reference-Cited by-同舟云学术

Genetic inactivation of p62 leads to accumulation of hyperphosphorylated tau and neurodegeneration

Published:2008-07 Issue:1 Volume:106 Page:107-120
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J Neurochem

Author:

Ramesh Babu J.,Lamar Seibenhener M.,Peng Junmin,Strom Anna-Lena,Kemppainen Robert,Cox Nancy,Zhu Haining,Wooten Michael C.,Diaz-Meco María T.,Moscat Jorge,Wooten Marie W.

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1471-4159.2008.05340.x/fullpdf

Reference58 articles.

1. Ubiquitin-mediated activation of TAK1 and IKK

2. Role of abnormally phosphorylated tau in the breakdown of microtubules in Alzheimer disease.

3. Hyperphosphorylation and aggregation of tau in mice expressing normal human tau isoforms

4. Neuronal and glial inclusions in frontotemporal dementia with or without motor neuron disease are immunopositive for p62

5. Sequestosome 1/p62 shuttles polyubiquitinated tau for proteasomal degradation

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