Altered microglial copper homeostasis in a mouse model of Alzheimer’s disease-Reference-Cited by-同舟云学术

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Altered microglial copper homeostasis in a mouse model of Alzheimer’s disease

Published:2010-09 Issue:6 Volume:114 Page:1630-1638
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:

Author:

Zheng Zhiqiang,White Carine,Lee Jaekwon,Peterson Troy S.,Bush Ashley I.,Sun Grace Y.,Weisman Gary A.,Petris Michael J.

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1471-4159.2010.06888.x/fullpdf

Reference61 articles.

1. Up-regulation of the inflammatory cytokines IFN-gamma and IL-12 and down-regulation of IL-4 in cerebral cortex regions of APP(SWE) transgenic mice;Abbas;J. Neuroimmunol.,2002

2. Rapid restoration of cognition in Alzheimer’s transgenic mice with 8-hydroxy quinoline analogs is associated with decreased interstitial Abeta;Adlard;Neuron,2008

3. Dramatic aggregation of Alzheimer abeta by Cu(II) is induced by conditions representing physiological acidosis;Atwood;J. Biol. Chem.,1998

4. Characterization of copper interactions with alzheimer amyloid beta peptides: identification of an attomolar-affinity copper binding site on amyloid beta1-42;Atwood;J. Neurochem.,2000

5. Tyrosine gated electron transfer is key to the toxic mechanism of Alzheimer’s disease beta-amyloid;Barnham;FASEB J.,2004

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