BIOCHEMICAL CHANGES IN ?,?'-IMINODIPROPIONITRILE-TREATED RAT BRAIN AND PREVENTION OF TOXICITY BY ETHIONINE
Author:
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience,Biochemistry
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1471-4159.1969.tb06856.x/fullpdf
Reference40 articles.
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1. Motor unit function during evolution of proximal axonal swellings;Journal of the Neurological Sciences;1992-05
2. The protein synthesis inhibitor, anisomycin, causes exacerbation of the iminodipropionitrile-induced spasmodic dyskinetic syndrome in rats;Pharmacology Biochemistry and Behavior;1989-04
3. The iminodipropionitrile (IDPN)-induced dyskinetic syndrome: Behavioral and biochemical pharmacology;Neuroscience & Biobehavioral Reviews;1989-03
4. Behavioural effects of β,β′-iminodipropionitrile (IDPN) in the domestic fowl (Gallus domesticus);General Pharmacology: The Vascular System;1984-01
5. Quipazine exacerbation of a hyperkinetic syndrome: Involvement of brain dopamine and serotonin;Journal of Neuroscience Research;1977
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