Calpain activation is involved in early caspase-independent neurodegeneration in the hippocampus following status epilepticus-Reference-Cited by-同舟云学术

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Calpain activation is involved in early caspase-independent neurodegeneration in the hippocampus following status epilepticus

Published:2008-05 Issue:3 Volume:105 Page:666-676
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J Neurochem

Author:

Araújo Inês M.,Gil Joana M.,Carreira Bruno P.,Mohapel Paul,Petersen Åsa,Pinheiro Paulo S.,Soulet Denis,Bahr Ben A.,Brundin Patrik,Carvalho Caetana M.

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1471-4159.2007.05181.x/fullpdf

Reference47 articles.

1. Early calpain-mediated proteolysis following AMPA receptor activation compromises neuronal survival in cultured hippocampal neurons

2. Proteolysis of NR2B by calpain in the hippocampus of epileptic rats

3. Changes in calcium dynamics following the reversal of the sodium-calcium exchanger have a key role in AMPA receptor-mediated neurodegeneration via calpain activation in hippocampal neurons

4. Neuronal nitric oxide synthase proteolysis limits the involvement of nitric oxide in kainate-induced neurotoxicity in hippocampal neurons

5. Molecular mechanisms of calcium-dependent neurodegeneration in excitotoxicity

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