Valproic acid reduces brain damage induced by transient focal cerebral ischemia in rats: potential roles of histone deacetylase inhibition and heat shock protein induction
Author:
Affiliation:
1. Molecular Neurobiology Section, National Institute of Mental Health, National Institutes of Health, Bethesda, Maryland, USA
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience,Biochemistry
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/j.1471-4159.2004.02406.x
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1. Specific Caspase Pathways Are Activated in the Two Stages of Cerebral Infarction
2. Gene activation by histone and factor acetyltransferases
3. Opposing Actions of Phosphatidylinositol 3-Kinase and Glycogen Synthase Kinase-3β in the Regulation of HSF-1 Activity
4. Increased expression of endoplasmic reticulum stress proteins following chronic valproate treatment of rat C6 glioma cells
5. Treatment of Acute Ischemic Stroke
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