Molecular mechanisms involved in the adenosine A1 and A2A receptor-induced neuronal differentiation in neuroblastoma cells and striatal primary cultures-Reference-Cited by-同舟云学术

Molecular mechanisms involved in the adenosine A1 and A2A receptor-induced neuronal differentiation in neuroblastoma cells and striatal primary cultures

Published:2005-01 Issue:2 Volume:92 Page:337-348
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J Neurochem

Author:

Canals Meritxell,Angulo Ester,Casado Vicent,Canela Enric I.,Mallol Josefa,Vinals Francesc,Staines William,Tinner Barbro,Agnati Luigi,Fuxe Kjell,Ferre Sergi,Lluis Carmen,Franco Rafael

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1471-4159.2004.02856.x/fullpdf

Reference47 articles.

1. A1 Adenosine Receptors Accumulate in Neurodegenerative Structures in Alzheimer's Disease and Mediate Both Amyloid Precursor Protein Processing and Tau Phosphorylation and Translocation

2. Signaling via A2A adenosine receptor in four PC12 cell clones

3. Endogenously Produced Neurotrophins Regulate Survival and Differentiation of Cortical Progenitors via Distinct Signaling Pathways

4. The cytostatic- and differentiation-inducing effects of cyclopentenyl cytosine on neuroblastoma cell lines11Abbreviations: CPEC, cyclopentenyl cytosine; and CPECTP, cyclopentenyl cytosine-5′-triphosphate.

5. Stimulation of the ERK Pathway by GTP-loaded Rap1 Requires the Concomitant Activation of Ras, Protein Kinase C, and Protein Kinase A in Neuronal Cells

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