Tumor necrosis factor-α-induced neutral sphingomyelinase-2 modulates synaptic plasticity by controlling the membrane insertion of NMDA receptors-Reference-Cited by-同舟云学术

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Tumor necrosis factor-α-induced neutral sphingomyelinase-2 modulates synaptic plasticity by controlling the membrane insertion of NMDA receptors

Published:2009-06 Issue:5 Volume:109 Page:1237-1249
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:

Author:

Wheeler David,Knapp Edward,Bandaru Veera V. R.,Wang Yue,Knorr David,Poirier Christophe,Mattson Mark P.,Geiger Jonathan D.,Haughey Norman J.

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1471-4159.2009.06038.x/fullpdf

Reference52 articles.

1. Protection by cholesterol-extracting cyclodextrins: a role for N-methyl-D-aspartate receptor redistribution;Abulrob;J. Neurochem.,2005

2. A deletion in the gene encoding sphingomyelin phosphodiesterase 3 (Smpd3) results in osteogenesis and dentinogenesis imperfecta in the mouse;Aubin;Nat. Genet.,2005

3. Subunit-specific NMDA receptor trafficking to synapses;Barria;Neuron,2002

4. Control of synaptic strength by glial TNFalpha;Beattie;Science,2002

5. Increased phosphorylation and redistribution of NMDA receptors between synaptic lipid rafts and post-synaptic densities following transient global ischemia in the rat brain;Besshoh;J. Neurochem.,2005

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