p53-Dependent control of cell death by nicastrin: lack of requirement for presenilin-dependent γ-secretase complex-Reference-Cited by-同舟云学术

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p53-Dependent control of cell death by nicastrin: lack of requirement for presenilin-dependent γ-secretase complex

Published:2009-04 Issue:1 Volume:109 Page:225-237
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:

Author:

Pardossi-Piquard Raphaëlle,Dunys Julie,Giaime Emilie,Guillot-Sestier Marie-Victoire,St. George-Hyslop Peter,Checler Frédéric,Alves da Costa Cristine

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1471-4159.2009.05952.x/fullpdf

Reference51 articles.

1. Wild-type and mutated presenilins 2 trigger p53-dependent apoptosis and down-regulate presenilin 1 expression in HEK293 human cells and in murine neurons;Alves da Costa;Proc. Natl Acad. Sci. USA,2002

2. Alpha-synuclein lowers p53-dependent apoptotic response of neuronal cells. Abolishment by 6-hydroxydopamine and implication for Parkinson’s disease;Alves da Costa;J. Biol. Chem.,2002

3. The C-terminal fragment of presenilin 2 triggers p53-mediated staurosporine-induced apoptosis, a function independent of the presenilinase-derived N-terminal counterpart;Alves da Costa;J. Biol. Chem.,2003

4. Presenilin-directed inhibitors of gamma-secretase trigger caspase 3 activation in presenilin-expressing and presenilin-deficient cells;Alves da Costa;J. Neurochem.,2004

5. Presenilin-dependent gamma-secretase-mediated control of p53-associated cell death in Alzheimer’s disease;Alves da Costa;J. Neurosci.,2006

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2. Nanoscale organization of Nicastrin, the substrate receptor of the γ-secretase complex, as independent molecular domains;Molecular Brain;2021-10-13

3. The P53/microRNA network: A potential tumor suppressor with a role in anticancer therapy;Pharmacological Research;2020-10

4. Effect of nicastrin on hepatocellular carcinoma proliferation and apoptosis through PI3K/AKT signalling pathway modulation;Cancer Cell International;2020-03-24

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