Roles of protein kinase Cα isozyme in the regulation of oxidative stress and neuropeptide Y gene expression in phenylpropanolamine-mediated appetite suppression
Author:
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience,Biochemistry
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1471-4159.2009.05909.x/fullpdf
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4. Protein kinase C isoforms as therapeutic targets in nervous system disease states;Battaini;Pharmacol. Res.,2001
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1. Targeting oxidative stress in the hypothalamus: the effect of transcription factor STAT3 knockdown on endogenous antioxidants-mediated appetite control;Archives of Toxicology;2014-05-04
2. Effects of selenoprotein S on oxidative injury in human endothelial cells;Journal of Translational Medicine;2013-11-14
3. Inhibiting neuropeptide Y Y1 receptor modulates melanocortin receptor- and NF-κB-mediated feeding behavior in phenylpropanolamine-treated rats;Hormones and Behavior;2013-06
4. The Identification of Neuropeptide Y Receptor Subtype Involved in Phenylpropanolamine-induced Increase in Oxidative Stress and Appetite Suppression;NeuroMolecular Medicine;2012-11-20
5. Neuropeptide Y Y1 receptor knockdown can modify glutathione peroxidase and c-AMP response element-binding protein in phenylpropanolamine-treated rats;Archives of Toxicology;2012-10-04
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