Reversible Alterations of Cerebral ?-Aminobutyric Acid in Pyrithiamine-Treated Rats: Implications for the Pathogenesis of Wernicke's Encephalopathy
Author:
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience,Biochemistry
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1471-4159.1988.tb03090.x/fullpdf
Reference26 articles.
1. Low Energy Levels in Thiamine-Deficient Encephalopathy
2. THE EFFECTS OF THIAMINE DEFICIENCY ON THE METABOLISM OF THE BRAIN?I. OXIDATION OF VARIOUS SUBSTRATES IN VITRO BY THE LIVER AND BRAIN OF NORMAL AND PYRITHIAMINE-FED RATS
3. Neurotransmitter function in thiamine-deficiency encephalopathy
4. Cerebral thiamine-dependent enzyme changes in experimental Wernicke's encephalopathy
5. Cerebral aminoacids in portal-systemic encephalopathy: Lack of evidence for altered ?-aminobutyric acid (GABA) function
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1. Alteration in cholinesterases, γ-aminobutyric acid and serotonin level with respect to thiamine deficiency in Swiss mice;Turkish Journal of Biochemistry;2018-08-28
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