The alternative splicing of tau exon 10 and its regulatory proteins CLK2 and TRA2-BETA1 changes in sporadic Alzheimer's disease-Reference-Cited by-同舟云学术

The alternative splicing of tau exon 10 and its regulatory proteins CLK2 and TRA2-BETA1 changes in sporadic Alzheimer's disease

Published:2006-02 Issue:3 Volume:96 Page:635-644
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J Neurochem

Author:

Glatz Daniela C.,Rujescu Dan,Tang Yesheng,Berendt Frank J.,Hartmann Annette M.,Faltraco Frank,Rosenberg Carlyn,Hulette Christine,Jellinger Kurt,Hampel Harald,Riederer Peter,Moller Hans-J.,Andreadis Athena,Henkel Kerstin,Stamm Stefan

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1471-4159.2005.03552.x/fullpdf

Reference34 articles.

1. Tau gene alternative splicing: expression patterns, regulation and modulation of function in normal brain and neurodegenerative diseases

2. Expression of tau mRNA and soluble tau isoforms in affected and non-affected brain areas in Alzheimer's disease

3. Staging of alzheimer's disease-related neurofibrillary changes

4. Overexpression of four-repeat tau mRNA isoforms in progressive supranuclear palsy but not in Alzheimer's disease

5. tau Exon 10 Expression Involves a Bipartite Intron 10 Regulatory Sequence and Weak 5′ and 3′ Splice Sites

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