Capacitative Ca2+ entry is involved in regulating soluble amyloid precursor protein (sAPPalpha) release mediated by muscarinic acetylcholine receptor activation in neuroblastoma SH-SY5Y cells-Reference-Cited by-同舟云学术

Capacitative Ca2+ entry is involved in regulating soluble amyloid precursor protein (sAPPalpha) release mediated by muscarinic acetylcholine receptor activation in neuroblastoma SH-SY5Y cells

Published:2006-04 Issue:1 Volume:97 Page:245-254
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J Neurochem

Author:

Kim Jin Hyoung,Choi Sinkyu,Jung Ji-Eun,Roh Eun-Jihn,Kim Hwa-Jung

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1471-4159.2006.03734.x/fullpdf

Reference70 articles.

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3. TRPC1-mediated Inhibition of 1-Methyl-4-phenylpyridinium Ion Neurotoxicity in Human SH-SY5Y Neuroblastoma Cells

4. Stable Activation of Single Ca2+ Release-activated Ca2+ Channels in Divalent Cation-free Solutions

5. Calcium regulates processing of the Alzheimer amyloid protein precursor in a protein kinase C-independent manner.

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