Identification and characterization of PEBP as a calpain substrate
Author:
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience,Biochemistry
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1471-4159.2006.04160.x/fullpdf
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2. Postischemic Administration of AK275, a Calpain Inhibitor, Provides Substantial Protection against Focal Ischemic Brain Damage
3. Calpain as a novel target for treating acute neurodegenerative disorders
4. Impairment of the Ubiquitin-Proteasome System by Protein Aggregation
5. Src-mediated Tyrosine Phosphorylation of NR2 Subunits of N-Methyl-d-Aspartate Receptors Protects from Calpain-mediated Truncation of Their C-terminal Domains
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