Zebrafish lacking Alzheimer presenilin enhancer 2 (Pen-2) demonstrate excessive p53-dependent apoptosis and neuronal loss-Reference-Cited by-同舟云学术

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Zebrafish lacking Alzheimer presenilin enhancer 2 (Pen-2) demonstrate excessive p53-dependent apoptosis and neuronal loss

Published:2006-02-08 Issue:5 Volume:96 Page:1423-1440
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:

Author:

Campbell William A.,Yang Hongwei,Zetterberg Henrik,Baulac Stéphanie,Sears Jacqueline A.,Liu Tianming,Wong Stephen T. C.,Zhong Tao P.,Xia Weiming

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1471-4159.2006.03648.x/fullpdf

Reference88 articles.

1. Presenilin 1 protein directly interacts with Bcl-2;Alberici;J. Biol. Chem.,1999

2. Wild-type and mutated presenilins 2 trigger p53-dependent apoptosis and down-regulate presenilin 1 expression in HEK293 human cells and in murine neurons;Alves da Costa;Proc. Natl Acad. Sci. USA,2002

3. The C-terminal fragment of presenilin 2 triggers p53-mediated staurosporine-induced apoptosis, a function independent of the presenilinase-derived N-terminal counterpart;Alves da Costa;J. Biol. Chem.,2003

4. Behavioral alterations associated with apoptosis and down-regulation of presenilin 1 in the brains of p53-deficient mice;Amson;Proc. Natl Acad. Sci. USA,2000

5. The levels of mature glycosylated nicastrin are regulated and correlate with γ-secretase processing of amyloid β-precursor protein;Arawaka;J. Neurochem.,2002

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