Neural mitochondrial Ca2+capacity impairment precedes the onset of motor symptoms in G93A Cu/Zn-superoxide dismutase mutant mice-Reference-Cited by-同舟云学术

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Neural mitochondrial Ca2+capacity impairment precedes the onset of motor symptoms in G93A Cu/Zn-superoxide dismutase mutant mice

Published:2006-01-30 Issue:5 Volume:96 Page:1349-1361
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:

Author:

Damiano Maria,Starkov Anatoly A.,Petri Susanne,Kipiani Kathuna,Kiaei Mahmoud,Mattiazzi Marina,Flint Beal M.,Manfredi Giovanni

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1471-4159.2006.03619.x/fullpdf

Reference61 articles.

1. Calcium induced release of mitochondrial cytochrome c by different mechanisms selective for brain versus liver;Andreyev;Cell Death Differ.,1999

2. Mitochondrial dysfunction and oxidative damage in Alzheimer's and Parkinson's diseases and coenzyme Q10 as a potential treatment;Beal;J. Bioenerg. Biomembr.,2004

3. Early vacuolization and mitochondrial damage in motor neurons of FALS mice are not associated with apoptosis or with changes in cytochrome oxidase histochemical reactivity;Bendotti;J. Neurol. Sci.,2001

4. Superoxide dismutase 1 with mutations linked to familial amyotrophic lateral sclerosis possesses significant activity;Borchelt;Proc. Natl Acad. Sci. USA,1994

5. ALS-linked SOD1 mutant G85R mediates damage to astrocytes and promotes rapidly progressive disease with SOD1-containing inclusions;Bruijn;Neuron,1997

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