Inverse and distinct modulation of tau-dependent neurodegeneration by presenilin 1 and amyloid-? in cultured cortical neurons: evidence that tau phosphorylation is the limiting factor in amyloid-?-induced cell death-Reference-Cited by-同舟云学术

Inverse and distinct modulation of tau-dependent neurodegeneration by presenilin 1 and amyloid-? in cultured cortical neurons: evidence that tau phosphorylation is the limiting factor in amyloid-?-induced cell death

Published:2007-06 Issue:5 Volume:101 Page:1303-1315
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J Neurochem

Author:

Leschik Julia,Welzel Alfred,Weissmann Carina,Eckert Anne,Brandt Roland

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1471-4159.2006.04435.x/fullpdf

Reference56 articles.

1. Pro-apoptotic effect of presenilin 2 (PS2) overexpression is associated with down-regulation of Bcl-2 in cultured neurons

2. PS1 activates PI3K thus inhibiting GSK-3 activity and tau overphosphorylation: effects of FAD mutations

3. Time sequence of maturation of dystrophic neurites associated with Aβ deposits in APP/PS1 transgenic mice

4. Functional Interactions of Tau and their Relevance for Alzheimers Disease

5. Interaction of tau with the neural plasma membrane mediated by tau's amino-terminal projection domain.

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