Pathogenesis and drug response of iPSC-derived cardiomyocytes from two Brugada syndrome patients with different Na<sub>v</sub>1.5-subunit mutations-Reference-Cited by-同舟云学术

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Pathogenesis and drug response of iPSC-derived cardiomyocytes from two Brugada syndrome patients with different Na_v1.5-subunit mutations

Published:2021 Issue:5 Volume:35 Page:395
ISSN:1674-8301
Container-title:The Journal of Biomedical Research
language:
Short-container-title:J Biomed Res

Author:

Zhu Yue,Wang Linlin,Cui Chang,Qin Huiyuan,Chen Hongwu,Chen Shaojie,Lin Yongping,Cheng Hongyi,Jiang Xiaohong,Chen Minglong

Publisher

Journal of Biomedical Research

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Cited by 8 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Analysis of the role of Na<sub>v</sub>1.5 slow inactivation in the development of inherited cardiac pathology;Цитология;2024-06-05

2. Analysis of Slow Inactivation of Nav1.5 Channels in the Development of Hereditary Heart Pathology;Cell and Tissue Biology;2024-06

3. Biophysical mechanisms of myocardium sodium channelopathies;Pflügers Archiv - European Journal of Physiology;2024-03-01

4. Patient-specific iPSC-derived cardiomyocytes reveal aberrant activation of Wnt/β-catenin signaling in SCN5A-related Brugada syndrome;Stem Cell Research & Therapy;2023-09-08

5. Brugada Syndrome: More than a Monogenic Channelopathy;Biomedicines;2023-08-18

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