Do reactive oxygen species damage or protect the heart in ischemia and reperfusion? Analysis on experimental and clinical data

Author:

Maslov Leonid N.,Naryzhnaya Natalia V.,Sirotina Maria,Mukhomedzyanov Alexandr V.,Kurbatov Boris K.,Boshchenko Alla A.,Ma Huijie,Zhang Yi,Fu Feng,Pei Jianming,Azev Viacheslav N.,Pereverzev Vladimir A.

Publisher

Journal of Biomedical Research

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Reference107 articles.

1. Bilenko MV. Ischemic and reperfusion injury to organs[M]. Moscow: Meditsina, 1989: 368.

2. Meerson FZ. Pathogenesis and prevention of stress and ischemic injuries of the heart[M]. Moscow: Meditsina, 1984: 272.

3. Meerson FZ, Krasikov SI, Ustinova EE, et al. Prevention of heart damage by extreme physical stress and increasing its resistance to acute overstrain with the antioxidant ionol[J]. Kardiologiia, 1986, 26(4): 70–74.

4. Yellon DM, Downey JM. Preconditioning the myocardium: from cellular physiology to clinical cardiology[J]. Physiol Rev, 2003, 83(4): 1113–1151.

5. Krylatov AV, Maslov LN, Voronkov NS, et al. Reactive oxygen species as intracellular signaling molecules in the cardiovascular system[J]. Curr Cardiol Rev, 2018, 14(4): 290–300.

Cited by 3 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献

1. O-GlcNAcylation in ischemic diseases;Frontiers in Pharmacology;2024-05-09

2. Editorial commentary on the special issue of cardiovascular diseases;The Journal of Biomedical Research;2023

3. Editorial commentary on the special Issue of cardiovascular diseases;The Journal of Biomedical Research;2023

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