Transient middle cerebral artery occlusion and reperfusion alters inducible NOS expression within the ventrolateral medulla and modulates cardiovascular function during static exercise
Author:
Affiliation:
1. Department of Pharmaceutical Sciences, South College School of Pharmacy, 400 Goody’s Lane, Knoxville, TN 37922, USA.
2. Department of Pharmaceutical Sciences, Massachusetts College of Pharmacy and Health Sciences, Boston, MA 02115, USA.
Abstract
Publisher
Canadian Science Publishing
Subject
Physiology (medical),Pharmacology,General Medicine,Physiology
Link
http://www.nrcresearchpress.com/doi/pdf/10.1139/y11-064
Reference54 articles.
1. Ventrolateral medullary control of cardiovascular activity during muscle contraction
2. Endothelial NOS expression within the ventrolateral medulla can affect cardiovascular function during static exercise in stroked rats
3. Ally, A., and Maher, T.J. 2010. Nitric oxide: its influence on neurotransmission within the ventrolateral medulla and on cardiovascular responses during static exercise following stroke. In Recent advances in cardiovascular research: from sleep to exercise. Transworld Research Network, Kerala, India. pp. 155–178.
4. Cardiovascular responses and neurotransmission in the ventrolateral medulla during skeletal muscle contraction following transient middle cerebral artery occlusion and reperfusion
5. Molecular changes in nNOS protein expression within the ventrolateral medulla following transient focal ischemia affect cardiovascular functions
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5. Modulation of inducible nitric oxide synthase (iNOS) expression and cardiovascular responses during static exercise following iNOS antagonism within the ventrolateral medulla;Molecular and Cellular Biochemistry;2014-09-19
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