Long-Term Methylglyoxal Treatment Causes Endothelial Dysfunction of Rat Isolated Mesenteric Artery-Reference-Cited by-同舟云学术

Long-Term Methylglyoxal Treatment Causes Endothelial Dysfunction of Rat Isolated Mesenteric Artery

Published:2013 Issue:2 Volume:75 Page:151-157
ISSN:0916-7250
Container-title:Journal of Veterinary Medical Science
language:en
Short-container-title:The Journal of Veterinary Medical Science

Author:

MUKOHDA Masashi¹,MORITA Tomoka¹,OKADA Muneyoshi¹,HARA Yukio¹,YAMAWAKI Hideyuki¹

Affiliation:

1. Laboratory of Veterinary Pharmacology, School of Veterinary Medicine, Kitasato University, Aomori 034–8628, Japan

Publisher

Japanese Society of Veterinary Science

Subject

General Veterinary

Link

https://www.jstage.jst.go.jp/article/jvms/75/2/75_12-0345/_pdf

Reference28 articles.

1. 1. Ahmed, M. U., Brinkmann Frye, E., Degenhardt, T. P., Thorpe, S. R. and Baynes, J. W. 1997. N-epsilon-(carboxyethyl)lysine, a product of the chemical modification of proteins by methylglyoxal, increases with age in human lens proteins. Biochem. J. 324565–570.

2. Role of Phosphodiesterase 3 in NO/cGMP-Mediated Antiinflammatory Effects in Vascular Smooth Muscle Cells

3. Hyperglycaemia-induced impairment of endothelium-dependent vasorelaxation in rat mesenteric arteries is mediated by intracellular methylglyoxal levels in a pathway dependent on oxidative stress

4. Oxidative stress and aging: Is methylglyoxal the hidden enemy?This review is one of a selection of papers published in a Special Issue on Oxidative Stress in Health and Disease.

5. Methylglyoxal scavengers attenuate endothelial dysfunction induced by methylglyoxal and high concentrations of glucose

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