Female Infertility in grt Mice is Caused by Thyroid Hormone Deficiency, not by Insufficient TPST2 Activity in the Reproductive Organs
Author:
Affiliation:
1. Laboratory of Laboratory Animal Science and Medicine, Department of Disease Control, Graduate School of Veterinary Medicine, Hokkaido University
Publisher
Japanese Society of Veterinary Science
Subject
General Veterinary
Link
http://www.jstage.jst.go.jp/article/jvms/70/10/70_10_1043/_pdf
Reference42 articles.
1. 1. Abel, M. H., Wootton, A. N., Wilkins, V., Huhtaniemi, I., Knight, P. G. and Charlton, H. M. 2000. The effect of a null mutation in the follicle-stimulating hormone receptor gene on mouse reproduction. Endocrinology 141: 1795-1803.
2. 2. Amendola, E., De Luca, P., Macchia, P. E., Terracciano, D., Rosica, A., Chiappetta, G., Kimura, S., Mansouri, A., Affuso, A., Arra, C., Macchia, V., Di Lauro, R. and De Felice, M. 2005. A mouse model demonstrates a multigenic origin of congenital hypothyroidism. Endocrinology 146: 5038-5047.
3. 3. Armada-Dias, L., Carvalho, J. J., Breitenbach, M. M., Franci, C. R. and Moura, E. G. 2001. Is the infertility in hypothyroidism mainly due to ovarian or pituitary functional changes? Braz. J. Med. Biol. Res. 34: 1209-1215.
4. Structural Differences in the Hinge Region of the Glycoprotein Hormone Receptors: Evidence from the Sulfated Tyrosine Residues
5. Targeted Disruption of Tyrosylprotein Sulfotransferase-2, an Enzyme That Catalyzes Post-translational Protein Tyrosine O-Sulfation, Causes Male Infertility
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