Apolipoprotein Gene Polymorphisms as Cause of Cholesterol QTLs in Mice
Author:
Affiliation:
1. Department of Molecular Biology and Immunology, National Institute of Agrobiological Sciences
Publisher
Japanese Society of Veterinary Science
Subject
General Veterinary
Link
https://www.jstage.jst.go.jp/article/jvms/67/6/67_6_583/_pdf
Reference36 articles.
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2. 2. Anunciad, R.V., Ohno, T., Mori, M., Ishikawa, A., Tanaka, S., Horio, F., Nishimura, M. and Namikawa, T. 2000. Distribution of body weight, blood insulin and lipid levels in the SMXA recombinant inbred strains and the QTL analysis. Exp. Anim. 49: 217-224.
3. 3. Choudhary, M. and Laurie, C.C. 1991. Use of in vitro mutagenesis to analyze the molecular basis of the difference in Adh expression associated with the allozyme polymorphism in Drosophila melanogaster. Genetics 129: 481-488.
4. 4. Cohen, R.D., Castellani, L.W., Qiao, J.-H., Van Lenten, B.J., Lusis, A.J. and Reue, K. 1997. Reduced aortic lesions and elevated high density lipoprotein levels in transgenic mice overexpressing mouse apolipoprotein A-IV. J. Clin. Invest. 99: 1906-1916.
5. 5. Higuchi, K., Kitagawa, K., Naiki, H., Hanada, K., Hosokawa, M. and Takeda, T. 1991. Polymorphism of apolipoprotein A-II (apoA-II) among inbred strains of mice. Biochem. J. 279: 427-433.
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