Mitigation of Pyrexia by a Th-1-Biased IgG Subclass Response after Infection with Equine Herpesvirus Type 1 in Horses Pre-Immunized with Inactivated Vaccine-Reference-Cited by-同舟云学术

Mitigation of Pyrexia by a Th-1-Biased IgG Subclass Response after Infection with Equine Herpesvirus Type 1 in Horses Pre-Immunized with Inactivated Vaccine

Published:2012 Issue:6 Volume:74 Page:791-795
ISSN:0916-7250
Container-title:Journal of Veterinary Medical Science
language:en
Short-container-title:The Journal of Veterinary Medical Science

Author:

BANNAI Hiroshi¹,TSUJIMURA Koji¹,KONDO Takashi¹,NEMOTO Manabu¹,YAMANAKA Takashi¹,SUGIURA Takeo¹,KATO Tomohiro²,MAEDA Ken³,MATSUMURA Tomio¹

Affiliation:

1. Epizootic Research Center, Equine Research Institute, Japan Racing Association, 1400-4 Shiba, Shimotsuke, Tochigi 329-0412, Japan

2. Racehorse Clinic, Ritto Training Center, Japan Racing Association, 1028 Misono, Ritto, Shiga 520-3085, Japan

3. Department of Veterinary Microbiology, Faculty of Agriculture, Yamaguchi University, 1677-1 Yoshida, Yamaguchi 753-8515, Japan

Publisher

Japanese Society of Veterinary Science

Subject

General Veterinary

Link

https://www.jstage.jst.go.jp/article/jvms/74/6/74_11-0449/_pdf

Reference29 articles.

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2. 2. Allen, G. P., Kydd, J. H., Slater, J. D. and Smith, K. C. 1999. Advances in understanding of the pathogenesis, epidemiology and immunological control of equine herpesvirus abortion. pp. 129-146. In: Equine Infectious Diseases VIII (Wernery, U., Wade, J. F., Mumford, J. A. and Kaaden, O. R. eds), R&W Publications, Newmarket.

3. 3. Allen, G., Yeargan, M., Costa, L. R. and Cross, R. 1995. Major histocompatibility complex class I-restricted cytotoxic T-lymphocyte responses in horses infected with equine herpesvirus 1. J. Virol. 69: 606-612.

4. Induction of a Th-1-Biased IgG Subclass Response against Equine Herpesvirus Type 1 in Horses Previously Infected with Type 4 Virus

5. The mucosal humoral immune response of the horse to infective challenge and vaccination with Equine herpesvirus-1 antigens

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