Anatomic Modifications in the Enteric Nervous System of JF1 Mice with the Classic Piebald Mutation
Author:
Affiliation:
1. Laboratory of Laboratory Animal Science and Medicine, Graduate School of Veterinary Medicine, Hokkaido University
Publisher
Japanese Society of Veterinary Science
Subject
General Veterinary
Link
https://www.jstage.jst.go.jp/article/jvms/74/3/74_11-0447/_pdf
Reference13 articles.
1. 1. Cantrell, V.A., Owens, S.E., Chandler, R.L., Airey, D.C., Bradley, K.M. et al. 2004. Interactions between Sox10 and EdnrB modulate penetrance and severity of aganglionosis in the Sox10Dom mouse model of Hirschsprung disease. Hum. Mol. Genet. 19: 2289-2301.
2. 2. Ceccherini, I., Zhang, A.L., Matera, I., Yang, G., Devoto, M. et al. 1995. Interstitial deletion of the endothelin-B receptor gene in the spotting lethal (sl) rat. Hum. Mol. Genet. 4: 2089-2096.
3. 3. Dang, R., Torigoe, D., Suzuki, S., Kikkawa, Y., Moritoh, K. et al. 2011. Genetic background strongly modifies the severity of symptoms of hirschsprung disease, but not hearing loss in rats carrying ednrb mutations. PloS. One 6: e24086.
4. 4. Dunn, L.C. 1920 Independent Genes in Mice. Genetics 5: 344-361.
5. GFRα1-Deficient Mice Have Deficits in the Enteric Nervous System and Kidneys
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