Genetic Background Strongly Influences the Severity of Glomerulosclerosis in Mice
Author:
Affiliation:
1. Laboratory of Laboratory Animal Science and Medicine, Department of Disease Control, Graduate School of Veterinary Medicine, Hokkaido University
2. Section of Biological Safety Research, Chitose Laboratory, Japan Food Research Laboratories
Publisher
Japanese Society of Veterinary Science
Subject
General Veterinary
Link
http://www.jstage.jst.go.jp/article/jvms/72/10/72_10-0144/_pdf
Reference21 articles.
1. 1. Andrews, K. L., Mudd, J. L., Li, C. and Miner, J. H. 2002. Quantitative trait loci influence renal disease progression in a mouse model of Alport syndrome. Am. J. Pathol. 160: 721-730.
2. Deficiency of the tensin2 gene in the ICGN mouse: an animal model for congenital nephrotic syndrome
3. Actin up: regulation of podocyte structure and function by components of the actin cytoskeleton
4. 4. Fogo, A. B. 2007. Mechanisms of progression of chronic kidney disease. Pediatr. Nephrol. 22: 2011-2022.
5. 5. Guo, M., Ricardo, S. D., Deane, J. A., Shi, M., Cullen-McEwen, L. and Bertram, J. F. 2005. A stereological study of the renal glomerular vasculature in the db/db mouse model of diabetic nephropathy. J. Anat. 207: 813-821.
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