Burn-induced subepicardial injury in frog heart: a simple model mimicking ST segment changes in ischemic heart disease
Author:
Affiliation:
1. Department of Physiology I, Tohoku University Graduate School of Medicine, Seiryo-cho, Aoba-ku, Sendai, Miyagi 980–8575, Japan
Publisher
Japanese Society of Veterinary Science
Subject
General Veterinary
Link
https://www.jstage.jst.go.jp/article/jvms/78/2/78_15-0440/_pdf
Reference16 articles.
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2. 2. Fasciano, R. W. 2nd. and Tung, L. 1999. Factors governing mechanical stimulation in frog hearts. Am. J. Physiol. 277: H2311–H2320.
3. 3. Fuller, W., Parmar, V., Eaton, P., Bell, J. R. and Shattock, M. J. 2003. Cardiac ischemia causes inhibition of the Na/K ATPase by a labile cytosolic compound whose production is linked to oxidant stress. Cardiovasc. Res. 57: 1044–1051.
4. 4. Ginghina, C., Ungureanu, C., Vladaia, A., Popescu, B. A. and Jurcut, R. 2009. The electrocardiographic profile of patients with angina pectoris. J. Med. Life 2: 80–91.
5. 5. Irisawa, H. and Kobayashi, M. 1964. On the spontaneous activities of oyster myocardium caused by several inorganic ions in sucrose solution. Jpn. J. Physiol. 14: 165–176.
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