Avidity of Anti-β2-Glycoprotein I and Thrombosis or Pregnancy Loss in Patients with Antiphospholipid Syndrome

Author:

ČUČNIK S.,BOŽIČ B.,KVEDER T.,TOMŠIČ M.,ROZMAN B.

Publisher

Wiley

Subject

History and Philosophy of Science,General Biochemistry, Genetics and Molecular Biology,General Neuroscience

Reference22 articles.

1. The anti-beta2-glycoprotein I activity in human anti-phospholipid syndrome sera is due to monoreactive low-affinity autoantibodies directed to epitopes located on native beta2-glycoprotein I and preserved during species' evolution;Tincani;J. Immunol.,1996

2. Anti-beta 2-glycoprotein I autoantibodies from patients with the “antiphospholipid” syndrome bind to beta 2-glycoprotein I with low affinity: dimerization of beta 2-glycoprotein I induces a significant increase in anti-beta 2-glycoprotein I antibody affinity;Sheng;J. Immunol.,1998

3. Monovalent binding of autoantibodies to beta2-glycoprotein I, detected using surface plasmon resonance at low antigen density;Regnault;Br. J. Haematol.,2000

4. High avidity anti-β2-glycoprotein I antibodies in patients with antiphospholipid syndrome;Cucnik;Ann. Rheum. Dis.,2004

5. Distinguishing features of anti-β2 glycoprotein I antibodies between patients with leprosy and the antiphospholipid syndrome;Arvieux;Thromb. Haemost.,2002

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