Local Amplification of Glucocorticoids by 11beta-Hydroxysteroid Dehydrogenase Type 1 and Its Role in the Inflammatory Response-Reference-Cited by-同舟云学术

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Local Amplification of Glucocorticoids by 11beta-Hydroxysteroid Dehydrogenase Type 1 and Its Role in the Inflammatory Response

Published:2006-11-01 Issue:1 Volume:1088 Page:265-273
ISSN:0077-8923
Container-title:Annals of the New York Academy of Sciences
language:en
Short-container-title:Annals of the New York Academy of Sciences

Author:

CHAPMAN K. E,COUTINHO A.,GRAY M.,GILMOUR J. S,SAVILL J. S,SECKL J. R

Publisher

Wiley

Subject

History and Philosophy of Science,General Biochemistry, Genetics and Molecular Biology,General Neuroscience

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1196/annals.1366.030/fullpdf

Reference55 articles.

1. A blast from the past: clearance of apoptotic cells regulates immune responses

2. Phagocytosis of apoptotic neutrophils does not induce macrophage release of thromboxane B2

3. Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGF-beta, PGE2, and PAF.

4. Immunosuppressive effects of apoptotic cells

5. Homozygous C1q deficiency causes glomerulonephritis associated with multiple apoptotic bodies

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4. Thiram, an inhibitor of 11ß-hydroxysteroid dehydrogenase type 2, enhances the inhibitory effects of hydrocortisone in the treatment of osteosarcoma through Wnt/β-catenin pathway;BMC Pharmacology and Toxicology;2023-03-28

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