The Role of Endoplasmic Reticulum Stress in Nonimmune Diabetes: NOD.k iHEL, a Novel Model of β Cell Death-Reference-Cited by-同舟云学术

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The Role of Endoplasmic Reticulum Stress in Nonimmune Diabetes: NOD.k iHEL, a Novel Model of β Cell Death

Published:2003-11 Issue:1 Volume:1005 Page:178-183
ISSN:0077-8923
Container-title:Annals of the New York Academy of Sciences
language:en
Short-container-title:

Author:

SOCHA L.,SILVA D.,LESAGE S.,GOODNOW C.,PETROVSKY N.

Publisher

Wiley

Subject

History and Philosophy of Science,General Biochemistry, Genetics and Molecular Biology,General Neuroscience

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1196/annals.1288.022/fullpdf

Reference23 articles.

1. Failure to censor forbidden clones of CD4 T cells in autoimmune diabetes;Lesage;J. Exp. Med.,2002

2. The NOD mouse: recessive diabetogenic gene in the major histocompatibility complex;Hattori;Science,1986

3. MHC ligands and peptide motifs: first listing;Rammensee;Immunogenetics,1995

4. Inhibition of autoimmune diabetes in nonobese diabetic mice by transgenic restoration of H2-E MHC class II expression: additive, but unequal, involvement of multiple APC subtypes;Johnson;J. Immunol.,2001

5. Analysis of susceptibility of NOD mice to spontaneous and experimentally induced thyroiditis;Damotte;Eur. J. Immunol.,1997

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