Establishment of Native Insulin-Negative NOD Mice and the Methodology to Distinguish Specific Insulin Knockout Genotypes and a B:16 Alanine Preproinsulin Transgene-Reference-Cited by-同舟云学术

Establishment of Native Insulin-Negative NOD Mice and the Methodology to Distinguish Specific Insulin Knockout Genotypes and a B:16 Alanine Preproinsulin Transgene

Published:2004-12 Issue:1 Volume:1037 Page:193-198
ISSN:0077-8923
Container-title:Annals of the New York Academy of Sciences
language:en
Short-container-title:

Author:

NAKAYAMA MAKI,MORIYAMA HIROAKI,ABIRU NORIO,BABU SUNANDA R.,SIKORA KAMILA,LI MARCELLA,MIAO DONGMEI,HUTTON JOHN C.,ELLIOTT JOHN F.,EISENBARTH GEORGE S.

Publisher

Wiley

Subject

History and Philosophy of Science,General Biochemistry, Genetics and Molecular Biology,General Neuroscience

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1196/annals.1337.031/fullpdf

Reference7 articles.

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2. Identification of an MHC class I-restricted autoantigen in type 1 diabetes by screening an organ-specific cDNA library;Wong;Nat. Med.,1999

3. Evidence for a primary islet autoantigen (preproinsulin 1) for insulitis and diabetes in the NOD mouse;Moriyama;Proc. Natl. Acad. Sci. USA,2003

4. Immunological characterization and therapeutic activity of an altered-peptide ligand, NBI-6024, based on the immunodominant type 1 diabetes autoantigen insulin B-chain (9-23) peptide;Alleva;Diabetes,2002

5. Establishing insulin 1 and insulin 2 knockout congenic strains on NOD genetic background;Paronen;Ann. N.Y. Acad. Sci.,2003

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5. Priming and effector dependence on insulin B:9–23 peptide in NOD islet autoimmunity;Journal of Clinical Investigation;2007-07-02