Ubiquitin like protein FAT10 repressed cardiac fibrosis after myocardial ischemic via mediating degradation of Smad3 dependent on FAT10-proteasome system-Reference-Cited by-同舟云学术

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Ubiquitin like protein FAT10 repressed cardiac fibrosis after myocardial ischemic via mediating degradation of Smad3 dependent on FAT10-proteasome system

Published:2023 Issue:3 Volume:19 Page:881-896
ISSN:1449-2288
Container-title:International Journal of Biological Sciences
language:en
Short-container-title:Int. J. Biol. Sci.

Author:

Chen Chen,Li Xiaoqing,Zhou Tao,Su Yuhao,Yu Bodong,Jin Jiejing,Xie Jinyan,Shen Yang,Wan Rong,Hong Kui

Publisher

Ivyspring International Publisher

Subject

Cell Biology,Developmental Biology,Molecular Biology,Applied Microbiology and Biotechnology,Ecology, Evolution, Behavior and Systematics

Cited by 4 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Crosstalk of ubiquitin system and non-coding RNA in fibrosis;International Journal of Biological Sciences;2024

2. Tissue fibrosis induced by radiotherapy: current understanding of the molecular mechanisms, diagnosis and therapeutic advances;Journal of Translational Medicine;2023-10-09

3. (E)‐SIS3 suppressed osteosarcoma progression via promoting cell apoptosis, arresting cell cycle, and regulating the tumor immune microenvironment;Drug Development Research;2023-10-02

4. Targeting selective E3 ligase for healing cardiac fibrosis;Cell Chemical Biology;2023-10

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