Modulation of H4K16Ac levels reduces pro-fibrotic gene expression and mitigates lung fibrosis in aged mice-Reference-Cited by-同舟云学术

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Modulation of H4K16Ac levels reduces pro-fibrotic gene expression and mitigates lung fibrosis in aged mice

Published:2022 Issue:2 Volume:12 Page:530-541
ISSN:1838-7640
Container-title:Theranostics
language:en
Short-container-title:Theranostics

Author:

Zhang Xiangyu,Liu Hui,Zhou Jennifer Q,Krick Stefanie,Barnes Jarrod W,Thannickal Victor J,Sanders Yan Y

Publisher

Ivyspring International Publisher

Subject

Pharmacology, Toxicology and Pharmaceutics (miscellaneous),Medicine (miscellaneous)

Cited by 11 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Tanshinone IIA positively regulates the Keap1-Nrf2 system to alleviate pulmonary fibrosis via the sestrin2-sqstm1 signaling axis-mediated autophagy;Phytomedicine;2024-07

2. Fibroblast Diversity and Epigenetic Regulation in Cardiac Fibrosis;International Journal of Molecular Sciences;2024-05-30

3. O‐GlcNAc regulates anti‐fibrotic genes in lung fibroblasts through EZH2;Journal of Cellular and Molecular Medicine;2024-03-17

4. KAT8 is upregulated and recruited to the promoter of Atg8 by FOXO to induce H4 acetylation for autophagy under 20-hydroxyecdysone regulation;Journal of Biological Chemistry;2024-03

5. Metabolic memory: mechanisms and diseases;Signal Transduction and Targeted Therapy;2024-02-28

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