PLK1-mediated phosphorylation of β-catenin enhances its stability and transcriptional activity for extracellular matrix remodeling in metastatic NSCLC-Reference-Cited by-同舟云学术

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PLK1-mediated phosphorylation of β-catenin enhances its stability and transcriptional activity for extracellular matrix remodeling in metastatic NSCLC

Published:2023 Issue:3 Volume:13 Page:1198-1216
ISSN:1838-7640
Container-title:Theranostics
language:en
Short-container-title:Theranostics

Author:

Kim Da-Eun,Shin Sol-Bi,Kim Chang-Hyeon,Kim Yeo-Bin,Oh Hyun-Ji,Yim Hyungshin

Publisher

Ivyspring International Publisher

Subject

Pharmacology, Toxicology and Pharmaceutics (miscellaneous),Medicine (miscellaneous)

Cited by 8 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Phosphorylation of LZTS2 by PLK1 activates the Wnt pathway;Cellular Signalling;2024-08

2. Disintegration of Cav-1/β-catenin complex attenuates neuronal death after ischemia–reperfusion injury by promoting β-catenin nuclear translocation;Molecular Biology Reports;2024-07-22

3. Proteasome activation is critical for cell death induced by inhibitors of polo-like kinase 1 (PLK1) in multiple cancers;European Journal of Pharmacology;2024-06

4. Telocinobufagin, a PLK1 suppressor that inhibits tumor growth and metastasis by modulating CDC25c and CTCF in HNSCC cells;Phytomedicine;2024-05

5. Vitamin A Status Modulates Epithelial Mesenchymal Transition in the Lung: The Role of Furin;Nutrients;2024-04-15

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