The Systemic Amyloid Precursor Transthyretin (TTR) Behaves as a Neuronal Stress Protein Regulated by HSF1 in SH-SY5Y Human Neuroblastoma Cells and APP23 Alzheimer's Disease Model Mice
Author:
Publisher
Society for Neuroscience
Subject
General Neuroscience
Cited by 50 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. The Role of CSF Transthyretin in Human Alzheimer's Disease: Offense, Defense, or not so Innocent Bystander;Journal of Integrative Neuroscience;2023-11-06
2. Mapping cellular response to destabilized transthyretin reveals cell- and amyloidogenic protein-specific signatures;Amyloid;2023-07-13
3. Multi-transcriptomics reveals brain cellular responses to peripheral infection in Alzheimer’s disease model mice;Cell Reports;2023-07
4. Human transthyretin gene expression is markedly increased in repair Schwann cells in an in vitro model of hereditary transthyretin amyloidosis;Neurochemistry International;2023-03
5. Stress-responsive regulation of extracellular proteostasis;Journal of Cell Biology;2022-02-22
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