Huntingtin Modulates Transcription, Occupies Gene Promoters In Vivo, and Binds Directly to DNA in a Polyglutamine-Dependent Manner-Reference-Cited by-同舟云学术

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Huntingtin Modulates Transcription, Occupies Gene Promoters In Vivo, and Binds Directly to DNA in a Polyglutamine-Dependent Manner

Published:2008-10-15 Issue:42 Volume:28 Page:10720-10733
ISSN:0270-6474
Container-title:Journal of Neuroscience
language:en
Short-container-title:Journal of Neuroscience

Author:

Benn C. L.,Sun T.,Sadri-Vakili G.,McFarland K. N.,DiRocco D. P.,Yohrling G. J.,Clark T. W.,Bouzou B.,Cha J.-H. J.

Publisher

Society for Neuroscience

Subject

General Neuroscience

Cited by 171 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. General loss of proteostasis links Huntington disease to Cockayne syndrome;Neurobiology of Disease;2024-09

2. Polyglutamine (PolyQ) Diseases: Navigating the Landscape of Neurodegeneration;ACS Chemical Neuroscience;2024-07-12

3. Beyond CAG Repeats: The Multifaceted Role of Genetics in Huntington Disease;Genes;2024-06-19

4. Huntingtin loss-of-function contributes to transcriptional deregulation in Huntington’s disease;2024-05-20

5. Mitochondria Modulating Therapeutic Approaches in the Management of Huntington’s Disease;Current Pharmacology Reports;2024-05-17

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