The Human γ-Globin TATA and CACCC Elements Have Key, Distinct Roles in Suppressing β-Globin Gene Expression in Embryonic/Fetal Development
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference36 articles.
1. A developmentally stable chromatin structure in the human beta-globin gene cluster.
2. The "beta-like-globin" gene domain in human erythroid cells.
3. An erythroid-specific, developmental-stage-independent enhancer far upstream of the human "beta-like globin" genes.
4. Position-independent, high-level expression of the human β-globin gene in transgenic mice
5. A deletion of the human beta-globin locus activation region causes a major alteration in chromatin structure and replication across the entire beta-globin locus.
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1. Native highland and lowland populations differ in .GAMMA.-globin gene promoter polymorphisms related to altered fetal hemoglobin levels and delayed fetal to adult globin switch after birth;Anthropological Science;2010
2. Fetal hemoglobin chemical inducers for treatment of hemoglobinopathies;Annals of Hematology;2008-11-15
3. Stress-induced mobility of OPHIO1 and OPHIO2, DNA transposons of the Dutch elm disease fungi;Fungal Genetics and Biology;2008-04
4. Transcriptional potential of the γ-globin gene is dependent on the CACCC box in a developmental stage-specific manner;Nucleic Acids Research;2006-08-12
5. Autonomous Silencing as Well as Competition Controls γ-Globin Gene Expression during Development;Molecular and Cellular Biology;2006-07
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