Antimitochondrial Autoantibodies in Pemphigus Vulgaris
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference75 articles.
1. A novel explanation for acantholysis in pemphigus vulgaris: The basal cell shrinkage hypothesis
2. Apoptolysis: a novel mechanism of skin blistering in pemphigus vulgaris linking the apoptotic pathways to basal cell shrinkage and suprabasal acantholysis
3. p38MAPK inhibition prevents disease in pemphigus vulgaris mice
4. An imbalance in Akt/mTOR is involved in the apoptotic and acantholytic processes in a mouse model of pemphigus vulgaris
5. Apoptotic mechanism in pemphigus autoimmunoglobulins-induced acantholysis—possible involvement of the EGF receptor
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2. Beyond the skin: B cells in pemphigus vulgaris, tolerance and treatment;British Journal of Dermatology;2024-03-20
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4. Non-coding regions of nuclear-DNA-encoded mitochondrial genes and intergenic sequences are targeted by autoantibodies in breast cancer;Frontiers in Genetics;2023-03-29
5. The Role of Non-Neuronal Acetylcholine in the Autoimmune Blistering Disease Pemphigus Vulgaris;Biology;2023-02-23
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