Calmodulin Activation Limits the Rate of KCNQ2 K+ Channel Exit from the Endoplasmic Reticulum-Reference-Cited by-同舟云学术

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Calmodulin Activation Limits the Rate of KCNQ2 K+ Channel Exit from the Endoplasmic Reticulum

Published:2009-07 Issue:31 Volume:284 Page:20668-20675
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Alaimo Alessandro,Gómez-Posada Juan Camilo,Aivar Paloma,Etxeberría Ainhoa,Rodriguez-Alfaro Jose Angel,Areso Pilar,Villarroel Alvaro

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference18 articles.

1. KCNQ2 and KCNQ3 Potassium Channel Subunits: Molecular Correlates of the M-Channel

2. Neuronal KCNQ potassium channels:physislogy and role in disease

3. The C-terminus of Kv7 channels: a multifunctional module

4. The Identification and Characterization of a Noncontinuous Calmodulin-binding Site in Noninactivating Voltage-dependent KCNQ Potassium Channels

5. Calmodulin as an Ion Channel Subunit

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1. Emerging mechanisms involving brain Kv7 channel in the pathogenesis of hypertension;Biochemical Pharmacology;2022-12

2. Kv Channel Ancillary Subunits: Where Do We Go from Here?;Physiology;2022-09-01

3. Molecular cloning and functional characterization of KCNQ1 in shell biomineralisation of pearl oyster Pinctada fucata martensii;Gene;2022-05

4. An epilepsy-causing mutation leads to co-translational misfolding of the Kv7.2 channel;BMC Biology;2021-05-21

5. The S2–S3 Loop of Kv7.4 Channels Is Essential for Calmodulin Regulation of Channel Activation;Frontiers in Physiology;2021-01-20

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