The Homocysteine-inducible Endoplasmic Reticulum Stress Protein Counteracts Calcium Store Depletion and Induction of CCAAT Enhancer-binding Protein Homologous Protein in a Neurotoxin Model of Parkinson Disease-Reference-Cited by-同舟云学术

The Homocysteine-inducible Endoplasmic Reticulum Stress Protein Counteracts Calcium Store Depletion and Induction of CCAAT Enhancer-binding Protein Homologous Protein in a Neurotoxin Model of Parkinson Disease

Published:2009-07 Issue:27 Volume:284 Page:18323-18333
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Chigurupati Srinivasulu,Wei Zelan,Belal Cherine,Vandermey Myriam,Kyriazis George A.,Arumugam Thiruma V.,Chan Sic L.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference58 articles.

1. Molecular Pathways of Neurodegeneration in Parkinson's Disease

2. THE BIOCHEMISTRY OF PARKINSON'S DISEASE

3. Failure of the ubiquitin–proteasome system in Parkinson's disease

4. ER Degradation of a Misfolded Luminal Protein by the Cytosolic Ubiquitin-Proteasome Pathway

5. Cellular response to endoplasmic reticulum stress: a matter of life or death

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