Ca2+ Depletion and Inositol 1,4,5-Trisphosphate-evoked Activation of Ca2+ Entry in Single Guinea Pig Hepatocytes
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference34 articles.
1. What drives calcium entry during [Ca2+]ioscillations? – challenging the capacitative model
2. Store depletion and calcium influx
3. The N-Terminal Domain of the IP3 Receptor Gates Store-Operated hTrp3 Channels
4. Modulation of Ca2+ entry by polypeptides of the inositol 1,4,5-trisphosphate receptor (IP3R) that bind transient receptor potential (TRP): Evidence for roles of TRP and IP3R in store depletion-activated Ca2+ entry
5. Functional interaction between InsP3 receptors and store-operated Htrp3 channels
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1. STORE-OPERATED Ca2+CHANNELS AND MICRODOMAINS OF Ca2+IN LIVER CELLS;Clinical and Experimental Pharmacology and Physiology;2009-01
2. Ca2+-permeable channels in the hepatocyte plasma membrane and their roles in hepatocyte physiology;Biochimica et Biophysica Acta (BBA) - Molecular Cell Research;2008-05
3. Capacitative calcium entry and transient receptor potential canonical 6 expression control human hepatoma cell proliferation;Hepatology;2008
4. TRPV channels and modulation by hepatocyte growth factor/scatter factor in human hepatoblastoma (HepG2) cells;Cell Calcium;2004-07
5. Polarized Calcium and Calmodulin Signaling in Secretory Epithelia;Physiological Reviews;2002-01-07
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