Absence of the γ Subunit of the Skeletal Muscle Dihydropyridine Receptor Increases L-type Ca2+ Currents and Alters Channel Inactivation Properties-Reference-Cited by-同舟云学术

Absence of the γ Subunit of the Skeletal Muscle Dihydropyridine Receptor Increases L-type Ca2+ Currents and Alters Channel Inactivation Properties

Published:2000-05 Issue:19 Volume:275 Page:14476-14481
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Freise Doris,Held Brigitte,Wissenbach Ulrich,Pfeifer Alexander,Trost Claudia,Himmerkus Nina,Schweig Uli,Freichel Marc,Biel Martin,Hofmann Franz,Hoth Markus,Flockerzi Veit

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference44 articles.

1. Restoration of excitation—contraction coupling and slow calcium current in dysgenic muscle by dihydropyridine receptor complementary DNA

2. Function of a truncated dihydropyridine receptor as both voltage sensor and calcium channel

3. Voltage-dependent calcium channels: From structure to function

4. STRUCTURE AND FUNCTION OF VOLTAGE-GATED ION CHANNELS

5. Dual Function of the Voltage-Dependent Ca2+ Channel α2δ Subunit in Current Stimulation and Subunit Interaction

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