Vascular Endothelial Growth Factor (VEGF)-driven Actin-based Motility Is Mediated by VEGFR2 and Requires Concerted Activation of Stress-activated Protein Kinase 2 (SAPK2/p38) and Geldanamycin-sensitive Phosphorylation of Focal Adhesion Kinase-Reference-Cited by-同舟云学术

Vascular Endothelial Growth Factor (VEGF)-driven Actin-based Motility Is Mediated by VEGFR2 and Requires Concerted Activation of Stress-activated Protein Kinase 2 (SAPK2/p38) and Geldanamycin-sensitive Phosphorylation of Focal Adhesion Kinase

Published:2000-04 Issue:14 Volume:275 Page:10661-10672
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Rousseau Simon,Houle François,Kotanides Helen,Witte Larry,Waltenberger Johannes,Landry Jacques,Huot Jacques

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference81 articles.

1. Vascular Endothelial Growth Factor, a Potent and Selective Angiogenic Agent

2. The human gene for vascular endothelial growth factor. Multiple protein forms are encoded through alternative exon splicing

3. Indirect angiogenic cytokines upregulate VEGF and bFGF gene expression in vascular smooth muscle cells, whereas hypoxia upregulates VEGF expression only.

4. The Vascular Endothelial Growth Factor Receptor Flt-1 Mediates Biological Activities

5. Hypoxia Induces Vascular Endothelial Growth Factor in Cultured Human Endothelial Cells

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