The Receptor for Advanced Glycation End Products Is Induced by the Glycation Products Themselves and Tumor Necrosis Factor-α through Nuclear Factor-κB, and by 17β-Estradiol through Sp-1 in Human Vascular Endothelial Cells-Reference-Cited by-同舟云学术

The Receptor for Advanced Glycation End Products Is Induced by the Glycation Products Themselves and Tumor Necrosis Factor-α through Nuclear Factor-κB, and by 17β-Estradiol through Sp-1 in Human Vascular Endothelial Cells

Published:2000-08 Issue:33 Volume:275 Page:25781-25790
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Tanaka Nobushige,Yonekura Hideto,Yamagishi Sho-ichi,Fujimori Hideki,Yamamoto Yasuhiko,Yamamoto Hiroshi

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference58 articles.

1. Advanced Glycosylation End Products in Tissue and the Biochemical Basis of Diabetic Complications

2. Advanced Glycation End Products-driven Angiogenesis in Vitro

3. Advanced glycation endproducts inhibit prostacyclin production and induce plasminogen activator inhibitor-1 in human microvascular endothelial cells

4. Receptor-Mediated Toxicity to Pericytes of Advanced Glycosylation End Products: A Possible Mechanism of Pericyte Loss in Diabetic Microangiopathy

5. Retinal Vascular Patterns

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