Nuclear Localization of a Non-caspase Truncation Product of Atrophin-1, with an Expanded Polyglutamine Repeat, Increases Cellular Toxicity-Reference-Cited by-同舟云学术

Nuclear Localization of a Non-caspase Truncation Product of Atrophin-1, with an Expanded Polyglutamine Repeat, Increases Cellular Toxicity

Published:2003-04 Issue:15 Volume:278 Page:13047-13055
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Nucifora Frederick C.,Ellerby Lisa M.,Wellington Cheryl L.,Wood Jon D.,Herring William J.,Sawa Akira,Hayden Michael R.,Dawson Valina L.,Dawson Ted M.,Ross Christopher A.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

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1. Unusual form of cerebellar ataxia: Combined dentato-rubral and pallido-Luysian degeneration

2. Familial myoclonus epilepsy and choreoathetosis: Hereditary dentatorubral-pallidoluysian atrophy

3. Huntington Disease and the Related Disorder, Dentatorubral-Pallidoluysian Atrophy (DRPLA)

4. Unstable expansion of CAG repeat in hereditary dentatorubral–pallidoluysian atrophy (DRPLA)

5. Dentatorubral-pallidoluysian atrophy: Clinical features are closely related to unstable expansions of trinucleotide (CAG) repeat

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