Role of the JAK/STAT Pathway in the Regulation of Interleukin-8 Transcription by Oxidized Phospholipids in Vitro and in Atherosclerosis in Vivo-Reference-Cited by-同舟云学术

Role of the JAK/STAT Pathway in the Regulation of Interleukin-8 Transcription by Oxidized Phospholipids in Vitro and in Atherosclerosis in Vivo

Published:2007-10 Issue:43 Volume:282 Page:31460-31468
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Gharavi Nima M.,Alva Jackelyn A.,Mouillesseaux Kevin P.,Lai Chi,Yeh Michael,Yeung Winnie,Johnson Jaclyn,Szeto Wan Lam,Hong Longsheng,Fishbein Michael,Wei Lai,Pfeffer Lawrence M.,Berliner Judith A.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference44 articles.

1. A Role for Oxidized Phospholipids in Atherosclerosis

2. A leukocyte homologue of the IL-8 receptor CXCR-2 mediates the accumulation of macrophages in atherosclerotic lesions of LDL receptor-deficient mice.

3. Increased Transcription of IL-8 in Endothelial Cells Is Differentially Regulated by TNF-α and Oxidized Phospholipids

4. Oxidized Phospholipids Increase Interleukin 8 (IL-8) Synthesis by Activation of the c-src/Signal Transducers and Activators of Transcription (STAT)3 Pathway

5. Integrin β3-mediated Src activation regulates apoptosis in IEC-6 cells via Akt and STAT3

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