Ataxia Telangiectasia Mutated (ATM) Is Essential for DNA-PKcs Phosphorylations at the Thr-2609 Cluster upon DNA Double Strand Break-Reference-Cited by-同舟云学术

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Ataxia Telangiectasia Mutated (ATM) Is Essential for DNA-PKcs Phosphorylations at the Thr-2609 Cluster upon DNA Double Strand Break

Published:2007-03 Issue:9 Volume:282 Page:6582-6587
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Chen Benjamin P.C.,Uematsu Naoya,Kobayashi Junya,Lerenthal Yaniv,Krempler Andrea,Yajima Hirohiko,Löbrich Markus,Shiloh Yosef,Chen David J.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference31 articles.

1. Chromosomal stability and the DNA double-stranded break connection

2. DNA double-strand breaks: signaling, repair and the cancer connection

3. The cellular response to general and programmed DNA double strand breaks

4. DNA Double-Strand Break Repair: All's Well that Ends Well

5. Requirement for the Kinase Activity of Human DNA-Dependent Protein Kinase Catalytic Subunit in DNA Strand Break Rejoining

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1. Targeting the ATM pathway in cancer: Opportunities, challenges and personalized therapeutic strategies;Cancer Treatment Reviews;2024-09

2. Secrets of DNA-PKcs beyond DNA repair;npj Precision Oncology;2024-07-23

3. ATM-dependent Phosphorylation of Nemo SQ Motifs Is Dispensable for Nemo-mediated Gene Expression Changes in Response to DNA Double-Strand Breaks;The Journal of Immunology;2024-07-15

4. Trypanosoma cruzi infection induces DNA double-strand breaks and activates DNA damage response pathway in host epithelial cells;Scientific Reports;2024-03-04

5. A-T neurodegeneration and DNA damage-induced transcriptional stress;DNA Repair;2024-03

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