CDK5/FBW7-dependent ubiquitination and degradation of EZH2 inhibits pancreatic cancer cell migration and invasion-Reference-Cited by-同舟云学术

登录注册会员服务联系我们

CDK5/FBW7-dependent ubiquitination and degradation of EZH2 inhibits pancreatic cancer cell migration and invasion

Published:2017-04 Issue:15 Volume:292 Page:6269-6280
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Jin Xin,Yang Chong,Fan Ping,Xiao Jun,Zhang Wanli,Zhan Sudong,Liu Tao,Wang Dejie,Wu Heshui

Funder

National Natural Science Foundation of China

Natural Science Foundation of Jiangxi Province

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference35 articles.

1. Pancreatic cancer in the spotlight;Lancet Oncol,2014

2. Cancer statistics: current diagnosis and treatment of pancreatic cancer in Shanghai;Long;China Cancer Lett,2014

3. Cancer epigenetics reaches mainstream oncology;Rodríguez-Paredes;Nat. Med,2011

4. Covalent histone modifications: miswritten, misinterpreted and mis-erased in human cancers;Chi;Nat. Rev. Cancer,2010

5. KDM2B promotes pancreatic cancer via Polycomb-dependent and -independent transcriptional programs;Tzatsos;J. Clin. Invest,2013

Cited by 89 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Molecular insights and clinical implications for the tumor suppressor role of SCFFBXW7 E3 ubiquitin ligase;Biochimica et Biophysica Acta (BBA) - Reviews on Cancer;2024-09

2. PRMT6 facilitates EZH2 protein stability by inhibiting TRAF6-mediated ubiquitination degradation to promote glioblastoma cell invasion and migration;Cell Death & Disease;2024-07-23

3. PRMT6 facilitates EZH2 protein stability by inhibiting TRAF6-mediated ubiquitination degradation to promote glioblastoma cell invasion and migration;2024-05-14

4. MicroRNAs as the critical regulators of epithelial mesenchymal transition in pancreatic tumor cells;Heliyon;2024-05

5. Regulation of EZH2 protein stability: new mechanisms, roles in tumorigenesis, and roads to the clinic;eBioMedicine;2024-02

同舟云学术

1.学者识别学者识别

2.学术分析学术分析

3.人才评估人才评估

"同舟云学术"是以全球学者为主线，采集、加工和组织学术论文而形成的新型学术文献查询和分析系统，可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容，当前同舟云学术共收录了国内外主流学术期刊6万余种，收集的期刊论文及会议论文总量共计约1.5亿篇，并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询！咨询电话：010-8811{复制后删除}0370

www.globalauthorid.com

TOP

Copyright © 2019-2024 北京同舟云网络信息技术有限公司
京公网安备11010802033243号京ICP备18003416号-3