O-GlcNAcase Is an RNA Polymerase II Elongation Factor Coupled to Pausing Factors SPT5 and TIF1β-Reference-Cited by-同舟云学术

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O-GlcNAcase Is an RNA Polymerase II Elongation Factor Coupled to Pausing Factors SPT5 and TIF1β

Published:2016-10 Issue:43 Volume:291 Page:22703-22713
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Resto Melissa,Kim Bong-Hyun,Fernandez Alfonso G.,Abraham Brian J.,Zhao Keji,Lewis Brian A.

Funder

National Cancer Institute

National Heart, Lung, and Blood Institute

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference75 articles.

1. The RNA polymerase II molecule at the 5′ end of the uninduced hsp70 gene of D. melanogaster is transcriptionally engaged;Rougvie;Cell,1988

2. The block to transcriptional elongation within the human c-myc gene is determined in the promoter-proximal region;Krumm;Genes Dev,1992

3. Hold back of RNA polymerase II at the transcription start site mediates down-regulation of c-myc in vivo;Strobl;EMBO J,1992

4. c-Myc regulates transcriptional pause release;Rahl;Cell,2010

5. Defining the status of RNA polymerase at promoters;Core;Cell Rep,2012

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1. O-GlcNAcase regulates pluripotency states of human embryonic stem cells;Stem Cell Reports;2024-07

2. Concerted transformation of a hyper-paused transcription complex and its reinforcing protein;Nature Communications;2024-04-08

3. The role of O-GlcNAcylation in RNA polymerase II transcription;Journal of Biological Chemistry;2024-03

4. Self-assembly of promoter DNA and RNA Pol II machinery into transcriptionally active biomolecular condensates;Science Advances;2023-10-20

5. O-GlcNAc transferase promotes glioblastoma by modulating genes responsible for cell survival, invasion, and inflammation;Journal of Biological Chemistry;2023-10

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